|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
POTENTIATION OF ANTIHYPERTENSIVE DRUGS
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
POTENTIATION OF ANTIHYPERTENSIVE DRUGS
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
CO-ADMINISTRATION MAY INCREASE CHANCES OF HYPOKALEMIA
|
FRUSEMIDE MAY ADD TO OR POTENTIATE THE THERAPEUTIC OF OTHER ANTIHYPERTENSIVE DRUGS
|
DIURETICS CAUSED HYPOKALEMIA MAY ANTAGONISE THE EFFECTS OF LIGNOCAIN
|
POTENTIATION OF ANTIHYPERTENSIVE DRUGS
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS MAY CAUSE HYPERCALCAEMIA DUE TO DECREASED CALCIUM EXCRETION
|
THIAZIDE DIURETICS MAY CAUSE HYPERCALCAEMIA DUE TO DECREASED CALCIUM EXCRETION
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
THIAZIDE DIURETICS DECREASE ITS URINARY EXCRETION LEADING TO HYPERCALCEMIA
|
CO-ADMINISTRATION WITH OTHER ANTIHYPERTENSIVES MAY RESULT IN ADDITIVE EFFECT OR POTENTIATION
|
DICLOFENAC CAN INHIBIT THE ACTIVITY OF DIURETICS
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
MAY POTENTIATE ORTHOSTATIC HYPOTENSION
|
MAY POTENTIATE ORTHOSTATIC HYPOTENSION
|
DICLOFENAC CAN INHIBIT THE ACTIVITY OF DIURETICS
|
INCREASED RISK OF HYPOKALEMIA
|
FRUSEMIDE MAY ADD TO OR POTENTIATE THE THERAPEUTIC OF OTHER ANTIHYPERTENSIVE DRUGS
|
MAY POTENTIATE HYPOTENSIVE EFFECTS
|
NEUROMUSCULAR BLOCKING ACTION OF TUBOCURARINE LIKE DRUGS IS ENHANCED
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
|
CO-ADMINISTRATION WITH OTHER ANTIHYPERTENSIVES MAY RESULT IN ADDITIVE EFFECT OR POTENTIATION
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
THIAZIDES AND OTHER DIURETICS, TEND TO PRODUCE HYPERGLYCEMIA AND MAY LEAD TO LOSS OF GLYCEMIC CONTROL
|
HYPOGLYCEMIC EFFECT IS REDUCED BY THE DRUG
|
CO-ADMINISTRATION WITH OTHER ANTIHYPERTENSIVES MAY RESULT IN ADDITIVE EFFECT OR POTENTIATION
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
THIAZIDE DIURETICS TEND TO PRODUCE HYPERGLYCEMIA LEADING TO LOSS OF DIABETES CONTROL
|
POTENTIATION OF ANTIHYPERTENSIVE DRUGS
|
ANTICOAGULANT EFFECT IS REDUCED
|
INCREASED RISK OF HYPOKALEMIA
|
THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
|
CO-ADMINISTRATION MAY INCREASE CHANCES OF HYPOKALEMIA
|
THIAZIDE DIURETICS MAY CAUSE HYPERCALCAEMIA DUE TO DECREASED CALCIUM EXCRETION
|
INCREASED RISK OF HYPOKALEMIA
|
POTENTIATION OF ORTHOSTATIC HYPOTENSION MAY OCCUR
|
THIAZIDE DIURETICS CONCOMITANT USE MAY LEAD TO TOXIC BLOOD LEVELS OF THE DRUG
|
AMIODARONE SHOULD BE USED WITH CAUTION WITH DRUGS THAT INDUCE HYPOKALEMIA AND /OR HYPOMAGNESEMIA
|
POTENTIATION OF ANTIHYPERTENSIVE DRUGS
|
INCREASED RISK OF HYPOKALEMIA
|
NSAIDS IN SOME PATIENTS CAN REDUCE THE DIURETIC, NATRIURETIC & ANTIHYPERTENSIVE EFFECTS OF LOOP, POTASSIUM SPARING OR THIAZIDE DIURETICS
|
DICLOFENAC CAN INHIBIT THE ACTIVITY OF DIURETICS
|
THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
|
POTENTIATION OF ANTIHYPERTENSIVE DRUGS
|
NEUROMUSCULAR BLOCKING ACTION OF TUBOCURARINE LIKE DRUGS IS ENHANCED
|
CO-ADMINISTRATION WITH OTHER ANTIHYPERTENSIVES MAY RESULT IN ADDITIVE EFFECT OR POTENTIATION
|
POTENTIATION OF ANTIHYPERTENSIVE DRUGS
|
CO-ADMINISTRATION OF THIAZIDE DIURETICS AND NSAIDS AGENTS CAN REDUCE THE DIURETIC, NATIURETIC, AND ANTIHYPERTENSIVE EFFECTS
|
CONCURRENT USE MAY CAUSE / POTENTIATE HYPOKALEMIA WHICH COULD PREDISPOSE THE PATIENT TO CARDIAC DYSFUNCTION
|
DIURETICS CAUSED HYPOKALEMIA MAY ANTAGONISE THE EFFECTS OF LIGNOCAIN
|
POTENTIATION OF ANTIHYPERTENSIVE DRUGS
|
DIURETICS CAUSED HYPOKALEMIA MAY ANTAGONISE THE EFFECTS OF LIGNOCAIN
|
CO-ADMINISTRATION WITH OTHER ANTIHYPERTENSIVES MAY RESULT IN ADDITIVE EFFECT OR POTENTIATION
|
DIURETICS REDUCE THE RENAL CLEARANCE OF LITHIUM AND ADD A HIGH RISK OF LITHIUM TOXICITY
|
CO-ADMINISTRATION WITH OTHER ANTIHYPERTENSIVES MAY RESULT IN ADDITIVE EFFECT OR POTENTIATION
|
MAY POTENTIATE ORTHOSTATIC HYPOTENSION
|