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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE HYPOTENSIVE EFFECTS OF BETA BLOCKERS MAY BE DIMINISHED BY THE CONCOMITANT ADMINISTRATION OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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BARBITURATES INCREASE ITS METABOLISM LEADING TO REDUCED EFFICACY
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BARBITURATES INCREASE ITS METABOLISM LEADING TO REDUCED EFFICACY
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BARBITURATES INCREASE ITS METABOLISM LEADING TO REDUCED EFFICACY
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BARBITURATES INCREASE ITS METABOLISM LEADING TO REDUCED EFFICACY
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BARBITURATES INCREASE ITS METABOLISM LEADING TO REDUCED EFFICACY
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BARBITURATES INCREASE ITS METABOLISM LEADING TO REDUCED EFFICACY
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CONCOMITANT USE OF DILTIAZEM WITH BETA BLOCKERS MAY RESULT IN ADDITIVE EFFECT ON CARDIAC CONDUCTION ; I.V. DILTIAZEM AND I.V. BETA-BLOCKERS SHOULD NOT BE ADMINISTRATION TOGETHER OR IN CLOSE PROXIMITY
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RIFAMPICIN MAY ACCELERATES METABOLISM OF THE DRUG & MAY CAUSE DECREASED PLASMA CONCENTRATION
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CLONIDINE CO-ADMINISTRATION WITH AGENTS KNOWN TO AFFECT SINUS NODE FUNCTION OR A.V. NODAL CONDUCTION LIKE DIGOXIN,CALCIUM CHANNEL BLOCKER & BETABLOCKER MAY CAUSE BRADYCARDIA & A.V. BLOCK
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ADRENALINE RESPONSE MAY BE REDUCED IN PATIENTS ON LONG TERM BETABLOCKERS
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ADDITIVE EFFECT WITH BETABLOCKERS ON AV NODE CONDUCTION
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION
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BETABLOCKERS NOT ONLY BLOCK THE PULMONARY EFFECTS OF BETA-AGONISTS LIKE ALBUTEROL BUT MAY PRODUCE SEVERE BRONCHOSPASM IN ASTHMATICS
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BETABLOCKERS MAY INHIBIT THE EFFECT OF BETASTIMULANTS LIKE BAMBUTEROL, XANTHINE DERIVATIVES & VICE VERSA
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BETABLOCKERS MAY REDUCE THE EFFECTIVENESS OF TREATMENT WITH PARASYMPATHOMIMETICS IN MYASTHENIA PTS. IN PTS RECIEVING BETABLOCKERS IT HAS PROLONGED BRADYCARDIA EFFECT
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BETABLOCKERS MAY REDUCE THE EFFECTIVENESS OF TREATMENT WITH PARASYMPATHOMIMETICS IN MYASTHENIA PTS. IN PTS RECIEVING BETABLOCKERS IT HAS PROLONGED BRADYCARDIA EFFECT
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PROPAFENONE MAY INCREASE THE PLASMA CONCENTRATION OF THE DRUG
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INCREASED HYPOTENSIVE EFFECT IS SEEN
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THEOPHYLLIN PLASMA CONCENTRATION IS RAISED BY THE DRUG
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THEOPHYLLIN PLASMA CONCENTRATION IS RAISED BY THE DRUG
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THEOPHYLLIN PLASMA CONCENTRATION IS RAISED BY THE DRUG
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THEOPHYLLIN PLASMA CONCENTRATION IS RAISED BY THE DRUG
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METHYSERGIDE VASOCONSTRICTIVE EFFECTS ARE ENHANCED BY THE DRUG WHICH MAY LEAD TO ARTERIAL OCCLUSION
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CONCOMITANT USE MAY INCREASE RISK OF VASCULAR OCCLUSION
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CONCOMITANT USE MAY INCREASE RISK OF VASCULAR OCCLUSION
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CONCOMITANT USE MAY INCREASE RISK OF VASCULAR OCCLUSION
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CONCOMITANT USE MAY INCREASE RISK OF VASCULAR OCCLUSION
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CONCOMITANT USE MAY INCREASE RISK OF VASCULAR OCCLUSION
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CONCOMITANT USE MAY INCREASE RISK OF VASCULAR OCCLUSION
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BETABLOCKERS MAY LEAD TO ADDITIVE NEGATIVE INOTROPIC EFFECTS
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BETABLOCKERS MAY PRODUCE BRADYCARDIA & HYPOTENSION
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MAOIS CAN CAUSE SEVERE HYPOTENSION WHEN GIVEN CONCURRENTLY
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ANTACIDS CAN DELAY & DECREASE ABSORPTION
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ANTACIDS CAN DELAY & DECREASE ABSORPTION
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ANTACIDS CAN DELAY & DECREASE ABSORPTION
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ANTACIDS CAN DELAY & DECREASE ABSORPTION
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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CONCURRENT ADMINISTRATION MAY CAUSE BRADYCARDIA, MYOCARDIAL DEPRESSION OR A.V. BLOCK
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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BETABLOCKERS CONCOMITANT USE MAY CAUSE SEVERE HYPERTENSION SINCE BETABLOCKERS OPPOSE THE BETA ADRENERGIC ACTION OF SYMPATHOMIMETICS
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POSSIBLE POTENTIATION OF BRADYCARDIA, SINUS ARREST AND AV BLOCK
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CIMETIDINE INCREASES ITS PLASMA CONCENTRATION AND HENCE BIOAVAILABILITY
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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NSAIDS CONCURRENT ADMINISTRATION MAY DECREASE THE ANTIHYPERTENSIVE EFFECT OF THE DRUG
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