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RENAL CLEARANCE MEASUREMENT CAN NOT BE MADE WITH ACCURACY IN PATIENTS RECEIVING THIS DRUG DUE TO INTERFERENCE WITH CHEMICAL COLOR DEVELOPMENT ESSENTIAL TO ANALYTICAL PROCEDURES
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CO-ADMINISTRATION WITH ANTIPLATELET AGENTS MAY BE ASSOCIATED WITH AN INCREASE IN BLEEDING.
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CO-ADMINISTRATION WITH ANTIPLATELET AGENTS MAY BE ASSOCIATED WITH AN INCREASE IN BLEEDING.
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FOLIC ACID DEFICIENCY MAY PRECIPITATE WITH CONCURRENT USE
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ACETAZOLAMIDE POTENTIATES THE EFFECTS OF FOLIC ACID ANTAGONISTS
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PHENYTOIN SERUM LEVELS ARE INCREASED BY THE DRUG
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ANTIBACTERIALS MAY INTERFERE WITH PLATELET FUNCTION OR WITH BACTERIAL SYNTHESIS OF VIT K IN THE GUT & THUS HAVE ANTICOAGULANT EFFECT OF THEIR OWN
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ANTIBACTERIALS MAY INTERFERE WITH PLATELET FUNCTION OR WITH BACTERIAL SYNTHESIS OF VIT K IN THE GUT & THUS HAVE ANTICOAGULANT EFFECT OF THEIR OWN
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ANTIBACTERIALS MAY INTERFERE WITH PLATELET FUNCTION OR WITH BACTERIAL SYNTHESIS OF VIT K IN THE GUT & THUS HAVE ANTICOAGULANT EFFECT OF THEIR OWN
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ANTIBACTERIALS MAY INTERFERE WITH PLATELET FUNCTION OR WITH BACTERIAL SYNTHESIS OF VIT K IN THE GUT & THUS HAVE ANTICOAGULANT EFFECT OF THEIR OWN
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ANTIBACTERIALS MAY INTERFERE WITH PLATELET FUNCTION OR WITH BACTERIAL SYNTHESIS OF VIT K IN THE GUT & THUS HAVE ANTICOAGULANT EFFECT OF THEIR OWN
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ANTIBACTERIALS MAY INTERFERE WITH PLATELET FUNCTION OR WITH BACTERIAL SYNTHESIS OF VIT K IN THE GUT & THUS HAVE ANTICOAGULANT EFFECT OF THEIR OWN
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ANTIBACTERIALS MAY INTERFERE WITH PLATELET FUNCTION OR WITH BACTERIAL SYNTHESIS OF VIT K IN THE GUT & THUS HAVE ANTICOAGULANT EFFECT OF THEIR OWN
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ANTAGONISTIC ANTIMALARIAL EFFECT
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ANTAGONISTIC ANTIMALARIAL EFFECT
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ANTAGONISTIC ANTIMALARIAL EFFECT
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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HYPOGLYCEMIC EFFECT IS POTENTIATED
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POSSIBILITY OF INTERACTION WITH OTHER HIGHLY PROTEIN BOUND DRUGS LIKE NSAIDS SHOULD BE CONSIDERED
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POSSIBILITY OF INTERACTION WITH OTHER HIGHLY PROTEIN BOUND DRUGS LIKE NSAIDS SHOULD BE CONSIDERED
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PROBENECID INCREASES THE SERUM LEVELS OF THE DRUG
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POTENTIAL FOR SULFONYLUREA TOXICITY
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CHLOROPROCAIN METABOLITE ( PARA-AMINOBENZOIC ACID) INHIBITS THE ACTION OF SULPHONAMIDES, HENCE SHOULD NOT BE CO-ADMINISTERED
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